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[DOI] [PMC free article] [PubMed] [Google Scholar] 70.Larrue R., Fellah S., Van der Hauwaert C., Hennino M.F., Perrais M., Lionet A., Glowacki F., Pottier N., Cauffiez C
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Mechanistically, TMAO may contribute to electrophysiological dysfunction by activating the protein kinase R-like endoplasmic reticulum kinase (PERK)/IRE1α axis, leading to increased atrial remodeling and subsequent arrhythmogenesis [62]