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In spinal moto-neurons of newborn rats, ethanol decreased the frequency of NMDAR-and AMPAR-dependent postsynaptic electrical signals (so-called excitatory postsynaptic currents [mEPSCs]), suggesting that ethanol inhibited glutamate release into the synapse (Ziskind-Conhaim et al

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NLRP3 inflammasome, a classical indicator of pyroptosis, whose activation not only contributes to pyroptosis but also has inextricable effects on apoptosis, necroptosis, and ferroptosis (62)