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This hepcidin downregulation fosters increased expression of the divalent metal transporter 1 and ferroportin within the duodenum, culminating in enhanced intestinal iron absorption [782, 783]
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To this end, we monitored the response of NAPstar3b in HEK293 cells and in HEK293 cells with a CRISPR-Cas9-mediated disruption of GSR (encoding glutathione reductase)
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